"My main reason for applying to the University Scholars Program was to further my knowledge regarding research procedures and techniques. I hope to use these skills to conduct and interpret medical research in my prospective career as a physician. My goals for the academic year are to do well on my MCAT and maintain a high GPA."
Medicine, specifically autoimmune disorders, cancer, and epigenetics.
Academic and Other Awards
- University Scholars Program Scholarship (2011-2012)
- Florida Academic Scholar
- Dean's List
- Phi Gamma Delta AAA Scholar
- Alpha Epsilon Delta
- Phi Gamma Delta
Footprints Buddy and Support Program, Shands Hospital Volunteer, and Global Medical Training
Hobbies and Interests
- Tennis, flag football, piano, music, and traveling.
Serpin Protection on Liver Ischemia Injury of Mice
Ischemia–reperfusion (I/R) injury is the main cause of both primary graft dysfunction (occurring in 10%–30% of grafts) and primary nonfunction of liver allografts (occurring in 5% of grafts). The latter is responsible for 81% of retransplantations during the first week after surgery (Clavien et al. 1992; Jaeschke 1996). Therefore, minimizing the adverse effects of I/R injury could increase the number of suitable grafts and the number of patients who successfully recover from liver transplantation. Presently, the effective therapy for the I/R injury of the liver is limited. Our aim is to check the protective effects of the viral proteins Serp1, Serp2, NSP, and MT7 on the mouse liver I/R. Blood supply from the left lobe of the B6 mice liver is going to be blocked for 90 minutes and reopened for 24 hours. The liver will then be harvested. Before the blockage of blood supply to the liver, the protein needs to be administered to the mice by the tail vein. Immunochemistry staining, H/E staining, Western Blot, and apoptosis methods will all be used to check the cell invasion and proteins associated with the apoptosis pathway. Real Time PCR will be used to evaluate the expression of genes associated with the inflammation and apoptosis pathway. The IFNR-/-, Caspase-1-/-, and Apo E-/- mice will be used to explore the mechanism of Serp1, Serp2, NSP, and MT7 on liver I/R.